That painful bump at the back of the heel is rarely just a bony problem. We use diagnostic ultrasound to find which tissues are actually driving your pain, then treat the real cause.
Haglund's deformity is a bony enlargement of the back of the heel bone (calcaneus), at the posterosuperior corner just above where the Achilles tendon attaches. Because rigid-backed shoes press on this prominence, it picked up the nickname "pump bump", after the stiff court pumps that aggravate it.
The bony bump on its own is often painless. The pain usually comes from what sits between the bone and the tendon: the retrocalcaneal bursa (a small fluid-filled cushion that becomes inflamed when compressed) and the insertional Achilles tendon (which can degenerate where it meets the heel). When the bony prominence, the bursitis and insertional Achilles tendinopathy occur together, the combination is sometimes called Haglund's syndrome.
This distinction matters, because the most effective treatment targets the irritated soft tissue and the mechanical forces compressing it, not the bone itself. Finding a prominent heel shape on imaging does not automatically explain your pain, and surgery to reduce the bone is rarely the first or best answer.
Plenty of people have a prominent posterior heel and no pain at all. When the back of the heel does hurt, the pain generators are usually the retrocalcaneal bursa and the insertional Achilles tendon being squeezed between the bony prominence and the heel counter of a shoe.
That is why two patients with an identical-looking bump can need completely different treatment. One may have an inflamed bursa that settles quickly with offloading and a footwear change. Another may have established insertional Achilles tendinopathy that needs a structured loading programme and shockwave. Treating both the same way is how heel pain ends up dragging on for months.
Every back-of-heel assessment at Lower Limb Clinic includes in-clinic diagnostic ultrasound. This lets us see the insertional Achilles tendon, the retrocalcaneal and superficial bursae and the bone surface in real time, during your first appointment, rather than weeks later after a separate referral.
We look for tendon thickening, hypoechoic (degenerative) change at the insertion, calcification, bursal fluid and increased blood flow on Power Doppler. Neovascularisation is a marker of active tissue pathology and helps us judge how irritable the tendon is and how to pace your loading programme.
We map how force moves through your feet during standing and walking. A high-arched (cavus) foot, a stiff first ray or a tight calf complex all change how the Achilles insertion is loaded, and these patterns guide both orthotic prescription and your loading plan.
We assess ankle dorsiflexion (a tight calf is a key driver), heel alignment, tendon strength and the exact shoes you spend most time in. For Haglund's, footwear is often the single most modifiable factor, and identifying which shoes compress the prominence frequently produces fast relief.
Around 90% of back-of-heel pain settles with structured non-surgical care. What we recommend depends on whether the main problem is bursitis, insertional tendinopathy, or both, and on the mechanics driving it.
The fastest win for most patients. Switching to softer or open-backed heel counters, adjusting the heel tab and removing the compression on the prominence often reduces symptoms quickly.
For insertional tendinopathy, a graded heavy-slow or eccentric programme performed on a flat surface (avoiding the dropped-heel position that compresses the insertion) is the strongest evidence-based treatment.
Strong evidence for insertional Achilles tendinopathy that has not responded to loading alone. Shockwave promotes tissue remodelling and reduces pain over a typical course of three to six weekly sessions.
A small heel lift reduces tensile load through the Achilles insertion, and custom orthoses (manufactured in-house via Réalta Labs using SLS 3D printing) address the cavus or asymmetric loading seen on gait analysis.
Temporary reduction of the aggravating loads (hill running, sprinting, deep dorsiflexion) while symptoms settle, then a structured return to full activity rather than open-ended rest.
For the minority who fail at least six months of structured conservative care, we refer to a trusted foot and ankle surgeon for consideration of bony resection or tendon debridement, with a clear rationale rather than as a first step.
A note on injections: corticosteroid injection directly into the Achilles insertion is generally avoided because of the risk to the tendon. Where an injection has a role, it is targeted and ultrasound-guided, and always combined with a loading programme.
Haglund's deformity reflects the bony shape of the heel combined with the loads placed on it. Several factors raise the risk of it becoming painful.
A high-arched (cavus) foot tilts the heel and increases prominence of the posterosuperior calcaneus. A tight calf and reduced ankle dorsiflexion raise insertional load.
Rigid or high heel counters (court pumps, ski boots, football boots, stiff dress shoes) compress the prominence and the bursa. This is the most modifiable factor.
Rapid increases in running volume, hill work and sprinting load the Achilles insertion. Common in runners returning too quickly after a break.
Inflammatory arthropathies can drive insertional pain and enthesopathy, and should be considered when both heels are affected or symptoms are out of proportion.
Onset, aggravating shoes and activities, previous treatment and impact on training and work.
Heel, ankle and calf examination including dorsiflexion range and Achilles strength.
Scanning the insertional tendon and bursae, shown to you in real time.
Force mapping to identify the mechanics loading the Achilles insertion.
A clear diagnosis, footwear guidance and a personalised plan with realistic timelines.
Total appointment time is approximately 45 to 60 minutes. Most patients leave with a clear diagnosis, a footwear plan and treatment already underway.
The outlook is good with the right diagnosis and a consistent plan. Bursitis driven mainly by footwear can settle within a few weeks once the compression is removed. Established insertional Achilles tendinopathy is slower and typically improves over 12 to 24 weeks of progressive loading, often supported by shockwave.
The biggest factors in recovery time are how long symptoms have been present before treatment, footwear, calf flexibility and consistency with the loading programme. We track progress with repeat ultrasound so decisions are based on measurable tissue change, not guesswork.
Paul McMullan BSc(Hons) MSc FRCPSGlasg MRCPod: Lead clinician with nearly 15 years of specialist experience in lower limb musculoskeletal conditions. Currently completing an MSc in Podiatric Sports Medicine at Queen Mary University of London. Experienced in diagnostic ultrasound, shockwave therapy and biomechanical assessment.
Darren Costello BSc(Hons) MSc MRCPSGlasg PGCert: MSc Sports and Exercise Medicine, with a postgraduate certificate from Brunel University in lower limb musculoskeletal assessment and a specialist interest in tendinopathy and orthotic therapy.
We see the tendon and bursa, rather than guessing from the bump.
Bursitis, insertional tendinopathy or both are managed differently.
Using 3D printing for a precise heel lift and shell.
For insertional Achilles cases that need more than loading.
Clear conservative pathway first, with onward referral when needed.
MSc-qualified clinicians in lower limb musculoskeletal care.
Common questions about Haglund's Deformity at Lower Limb Clinic.
Get specialist Haglund's Deformity treatment at your nearest Belfast clinic
385 Lisburn Road, BT9 7EP
373 Ormeau Road, BT7 3GP
We serve patients from across Belfast and Northern Ireland including East Belfast, South Belfast, Lisburn, Bangor, Holywood, Newtownards, Dundonald, Carryduff, Hillsborough, and Comber.