Accurate diagnosis is everything with heel spurs. Most cause no pain at all — so if yours hurts, we find out exactly why and treat the real problem.
A heel spur (calcaneal spur) is a bony growth that forms on the underside or back of the heel bone. These calcium deposits develop gradually over months and years, usually in response to repetitive strain on the plantar fascia or Achilles tendon where they attach to the calcaneus.
Here's what most people don't realise: heel spurs are remarkably common in people with no pain at all. Research consistently shows that around 15–25% of the general population have plantar heel spurs visible on X-ray, yet the majority experience no symptoms whatsoever. This is a critical point, because it means that finding a heel spur on imaging doesn't automatically explain your pain.
There are two distinct types of heel spur, and the distinction matters for treatment:
Plantar heel spurs grow on the underside of the calcaneus, at the origin of the plantar fascia. These are the most common type and are frequently found alongside plantar fasciitis — though the relationship between the two is more complex than simply "the spur causes the pain." Current evidence suggests the spur is a consequence of chronic fascial strain rather than the primary pain generator.
Posterior heel spurs (also called dorsal calcaneal spurs or Haglund's deformity–associated spurs) develop at the back of the heel where the Achilles tendon inserts. These are associated with insertional Achilles tendinopathy and can cause pain when shoes press against the back of the heel.
This is perhaps the most important thing we explain to patients: in the vast majority of cases, removing or treating the spur itself is unnecessary. The pain comes from the soft tissue pathology surrounding the spur — inflamed plantar fascia, degenerative tendon tissue, fat pad atrophy, or nerve entrapment.
A landmark systematic review by Johal & Milner (2012) found no consistent correlation between the size of a heel spur and the severity of symptoms. Equally, surgical removal of heel spurs alone (without addressing the underlying fasciopathy) has a high recurrence rate. This is why our approach focuses on identifying and treating the true pain source rather than fixating on what appears on an X-ray.
Getting the diagnosis right determines whether your treatment will actually work. Treating plantar fasciitis with a stretching and loading programme is highly effective. Treating a calcaneal stress fracture with stretching is potentially harmful. Treating Baxter's nerve entrapment with shockwave therapy alone is unlikely to resolve it. This is why we use diagnostic ultrasound as a core part of every heel pain assessment.
Every heel pain assessment at Lower Limb Clinic includes in-clinic diagnostic ultrasound. This allows us to visualise the plantar fascia, Achilles tendon, fat pad, and surrounding structures in real time — during your first appointment, not weeks later after a separate imaging referral.
Normal plantar fascia thickness measures approximately 3–4mm on ultrasound. In plantar fasciitis, the fascia typically thickens to 5mm or beyond, often with areas of hypoechoic (darker) degeneration visible within the tissue. We can also identify fascial tears, bursitis, fat pad changes, and assess blood flow using Power Doppler imaging — increased vascularity (neovascularisation) is a marker of active tissue pathology and helps guide treatment decisions.
Ultrasound also identifies conditions that X-rays miss entirely: early stress reactions in the calcaneus, soft tissue masses, plantar fibromatosis (Ledderhose disease), and retrocalcaneal bursitis behind the heel.
We use in-floor pressure plate technology to map exactly how forces distribute through your feet during standing and walking. This reveals asymmetries and abnormal loading patterns that contribute to chronic heel pain — such as excessive medial heel loading, late-phase forefoot overload, or compensatory gait patterns you may not be aware of.
This data directly informs orthotic prescription. Rather than using a generic arch support, we design orthoses based on your actual force distribution, targeting the specific mechanical factors driving your symptoms.
A detailed assessment of foot and ankle range of motion, lower limb alignment, muscle strength, and functional movement. We assess dorsiflexion range at the ankle (tightness in the calf complex is one of the strongest risk factors for plantar fasciopathy), rearfoot motion, midfoot stability, and hip and knee alignment. This gives us the full biomechanical picture — not just what's happening at the heel, but why.
Our treatment approach is guided by diagnosis. What we recommend for plantar fasciitis with heel spur is different from what we recommend for fat pad atrophy, Baxter's neuropathy, or insertional Achilles tendinopathy.
For plantar fasciopathy, the strongest evidence supports progressive loading — beginning with isometric calf raises, progressing through isotonic loading, and building toward functional demands. The Rathleff protocol (high-load strength training) has shown superior outcomes to traditional stretching alone.
NICE-approved for recalcitrant heel pain. We typically deliver 3–6 sessions at weekly intervals. Shockwave promotes neovascularisation and tissue remodelling in degenerative fascia — essentially restarting the healing process in tissue stuck in a chronic degenerative cycle.
Designed using pressure plate data and 3D foot scanning, manufactured in-house via Réalta Labs using selective laser sintering (SLS) 3D printing. Incorporating targeted heel cushioning, calibrated arch support, and forefoot modifications based on gait analysis.
For severe, acute plantar fasciopathy that hasn't responded to conservative measures. Performed under real-time ultrasound guidance. Injection creates a window for rehabilitation — we always combine it with a structured loading programme.
For fat pad atrophy (common in patients over 50), cushioning and offloading strategies are more appropriate than loading programmes. Silicone heel cups, viscoelastic cushioning in orthotics, and footwear modification can dramatically reduce symptoms.
Specific, evidence-based footwear advice including heel-to-toe drop, sole stiffness, cushioning properties, and fit. For posterior heel spurs, shoe modification or heel-tab adjustment can eliminate the friction driving symptoms.
Heel spurs develop in response to chronic mechanical stress. The bone remodels and deposits calcium at the point of maximum fascial or tendinous tension — a process that occurs over months to years.
Age over 40 (fat pad naturally loses thickness and elasticity). High body mass index increases compressive load through the heel with every step.
Prolonged standing or walking on hard surfaces. Rapid increases in training volume or intensity in runners and athletes.
Tight calf muscles (reduced ankle dorsiflexion below 10 degrees is strongly associated with plantar fasciopathy). Pes planus (flat feet) or pes cavus (high-arched feet) — both alter fascial loading patterns.
Inflammatory conditions such as psoriatic arthritis, reactive arthritis, or ankylosing spondylitis can drive enthesopathic spur formation.
Duration, aggravating factors, previous treatments, and impact on daily activities.
Foot, ankle, and lower limb examination including range of motion and muscle strength testing.
Scanning the plantar fascia, fat pad, and surrounding structures. Images shown to you in real time.
Force distribution mapping during walking to identify biomechanical contributors.
Clear explanation of your diagnosis with a personalised treatment plan, timelines, and expected outcomes.
Total appointment time: approximately 45–60 minutes. Most patients leave with a clear diagnosis and treatment already started — not a vague recommendation to "rest and see how it goes."
The prognosis for heel spur–related pain is excellent with appropriate treatment. Around 90% of plantar fasciopathy resolves with conservative (non-surgical) management. The key factors that influence recovery time include how long you've had symptoms before seeking treatment, accuracy of diagnosis, compliance with loading programmes, and addressing underlying biomechanical factors through orthotics and footwear.
Typical timelines: acute plantar fasciitis with early intervention — 6–12 weeks. Chronic plantar fasciopathy (over 6 months) — 12–24 weeks. Fat pad atrophy — ongoing management. Insertional Achilles tendinopathy with posterior heel spurs — 12–24 weeks with progressive loading.
We monitor progress with repeat ultrasound scanning, tracking measurable changes in fascial thickness, echogenicity, and vascularity alongside your subjective pain scores. This means treatment decisions are based on objective tissue changes, not guesswork.
Paul McMullan BSc(Hons) MSc FRCPSGlasg MRCPod — Lead clinician with nearly 15 years of specialist experience in lower limb musculoskeletal conditions. Currently completing an MSc in Podiatric Sports Medicine at Queen Mary University of London. Fellow of the Royal College of Physicians and Surgeons of Glasgow. Experienced in diagnostic ultrasound, injection therapy, shockwave therapy, and biomechanical assessment.
Darren Costello BSc(Hons) MSc PGCert — MSc Sports and Exercise Medicine, postgraduate certificate from Brunel University in lower limb musculoskeletal assessment. Specialist interest in biomechanical analysis and orthotic therapy for chronic heel conditions.
No separate imaging referral needed.
Not just what patients describe.
Using 3D printing technology for precision fit.
For chronic cases that haven't responded to standard treatment.
With specialist training in musculoskeletal lower limb conditions.
Fellowship of the Royal College of Physicians and Surgeons of Glasgow.
Common questions about Heel Spurs at Lower Limb Clinic.
Get Heel Spurs at our clinic, conveniently located in East Belfast
385 Lisburn Road, BT9 7EP
373 Ormeau Road, BT7 3GP
1 Shimna Road, BT33 0AS
We serve patients from across Belfast and Northern Ireland including East Belfast, South Belfast, Lisburn, Bangor, Holywood, Newtownards, Dundonald, Carryduff, Hillsborough, and Comber.
Get Heel Spurs at our clinic, conveniently located in East Belfast
385 Lisburn Road, BT9 7EP
373 Ormeau Road, BT7 3GP
1 Shimna Road, BT33 0AS
We serve patients from across Belfast and Northern Ireland including East Belfast, South Belfast, Lisburn, Bangor, Holywood, Newtownards, Dundonald, Carryduff, Hillsborough, and Comber.